Secondhand Smoke
Treated as a matter of degree rather than kind. Breathing ambient tobacco smoke deposits tars throughout the body just as direct smoking does, producing a diffuse, patchy accumulation pattern that iridology distinguishes from the concentrated lung deposits of active smokers.
Secondhand cigarette smoke, in Aajonus's framework, is not a substantially different phenomenon from direct smoking. It is a matter of degree rather than kind. Anyone who breathes air contaminated by tobacco combustion is taking smoke tars and carbon monoxides into the body, and the body must process and store those compounds just as it would in an active smoker. The lungs do not distinguish between smoke drawn deliberately through a cigarette and smoke inhaled passively from the air of a room, a workshop, or a vehicle. Particles are airborne, they are breathed in, and the body has to deal with them, period.
What Aajonus observed in iridology and hands-on assessment was that secondhand smokers show a recognizable but distinct pattern of tar accumulation compared to people who smoked directly. Active smokers tend to have tars concentrated in particular spots, especially around the lungs and kidneys. Secondhand smokers, by contrast, have tars distributed in patches throughout the system rather than concentrated in the respiratory organs. He described it as looking like the tars are scattered all over in particular spots rather than deeply embedded in the lung tissue the way they are in lifelong smokers. The pattern is diffuse rather than focal.
Aajonus was direct about this equivalence when reading bodies: "If they smoked and you were around them, you were a smoker, so you smoked for all of your childhood." This framing was consistent across his seminars. Being in a smoke-filled environment for years of childhood, or working daily in a shop where other people smoked, accumulated tars in the body at a level that registered on iris analysis as nearly indistinguishable from direct smoking.
Secondhand Smoke Body Accumulation
Smoke tars enter through respiration and are carried into the lungs, the bronchials, the lymphatic system, and from there throughout the body. Aajonus described tars as essentially cooked fats, produced by combustion at temperatures between 500 and 1,250 degrees Fahrenheit depending on how forcefully smoke is drawn. These high-temperature combustion products behave chemically as transfatty acids. The body attempts to use them as fats but cannot metabolize them efficiently because they are too thick and structurally damaged. Over time, if they remain in the body without being dissolved and eliminated, they crystallize and harden in tissues, eventually clogging the kidneys, lungs, and other organs.
He stated that smoke is a diet of smoke tars in that it is part of the element that goes into the body, and the body has to deal with it and try to utilize it as a fat. The difficulty is that it is very thick. This applies equally whether the tars arrived via a cigarette held in the hand or via ambient air in a nightclub, a mechanic's shop, or a childhood home. The body does not categorize the source; it only processes the compound.
Aajonus described examining a person who worked as a mechanic in a shop where other workers smoked, and finding that his lung and chest tissue read almost identically to a direct smoker in iridology. He told this man directly: "You're smoking with them. If they're smoking, you're smoking." The person had never smoked personally. Yet the tars in his chest were so extensive that they did not look like secondhand smoke at all on first reading.
He also read a person who had grown up with parents who smoked, never smoking personally, and observed that the tar distribution matched what he associated with secondhand exposure: patched throughout the system rather than concentrated in the lungs alone. Some of the tar damage appeared to have reached emotional centers, with spots around the right and left middle lobes of the lung area, and there was a large area of tar congestion in the ascending colon.
The Crystallization Problem
Over years of accumulation, smoke tars in the body do not remain soft or fluid. Aajonus explained that they crystallize and harden into the tissues. He described this as becoming like plaque hardened into tissue, or like beef jerky, drying and leathering the cellular environment. In people who had been around smoke for many years without eliminating the tars, this hardening progressed to the point of causing what he described as signs of tumorous activity, dangerous fibroid development, and obstruction of lymphatic flow.
In one body reading he described a man's tars as having gone into "leather," and warned that the pattern resembled what he had seen in people with advanced lung cancer. The person in that case was 26 years old, had smoked directly for some years, and had used marijuana. Aajonus noted that marijuana tars are 16 to 20 times more concentrated than regular cigarette tars, making them especially prone to crystallization because they burn at between 1,200 and 1,800 degrees Fahrenheit and produce transfatty acids that bond together and harden rapidly.
Even for people who never smoked directly, long-term secondhand exposure could produce enough crystallized tar to cause serious tissue damage. The pattern he described for such individuals was not the dramatic lung concentrations seen in heavy direct smokers, but rather a distributed plaque throughout the system that could press into glands, organs, and neural tissue over time.
Stage and Nightclub Smoke Danger
Aajonus drew a sharp distinction between conventional tobacco smoke, whether inhaled directly or secondhand, and the synthetic smoke used in theatrical performances and nightclub environments. He considered the synthetic product substantially more dangerous than tobacco smoke, and he encountered multiple patients whose cancers he attributed directly to repeated exposure to this substance.
The material used in stage smoke productions, as he described it, is not dry ice or any natural compound. It is vaporized hydrogenated vegetable oil, which he characterized as vaporized liquid plastic. Breathing particleized, vaporized plastic oil, he said, regardless of claims that it is derived from vegetable sources, introduces a plastic poison into the respiratory and lymphatic systems. Hydrogenated oil is plastic; it does not matter that its base material was once a vegetable.
He stated flatly that he would not attend any theater or venue using these products, and that if they were activated during a performance he was attending, he would leave. He wore a mask during at least one performance where he went specifically to warn a musician friend about his exposure levels.
He described two cases in detail. The first was a nightclub singer who performed for years in environments heavy with stage smoke. This woman developed cancer that eventually destroyed her entire nose. Aajonus said that the cancer discharged all kinds of plastic particles throughout her sinuses and brain. She ultimately died of natural causes without a nose, having refused the surgical interventions doctors recommended. He said the doctors wanted to go in, but she declined. What she was discharging was particleized plastic that had accumulated from years of breathing vaporized hydrogenated oil.
The second case was Malcolm Gold, a guitar player and singer who spent a year touring with a production that used heavy stage smoke for two to three hours per performance. Aajonus attended one of his shows in Los Angeles and sat at the back wearing a mask. He told the musician he would have to stop performing in those conditions. Malcolm Gold finished the tour and then quit. Approximately three to ten months after quitting, a tumor appeared at the eye. Aajonus described it as cancer coming right out of the eye, and said the worst that could happen was losing the eye. Gold did not see a doctor. He applied lime juice and vinegar topically as directed by Aajonus. Aajonus showed photographs at workshops of the progression: the tumor appearing, growing, then shrinking and healing. He had used pineapple and vinegar on the neck area of the same patient to break down accumulated dead cells on one side, and noted the difference in skin texture on the treated versus untreated side.
He also mentioned a third patient, a woman who was a nightclub singer and who developed cancer that affected her nasal area. He said a bandage was placed over her nose, she could no longer be hired as a singer, and the case was connected directly to the new kind of smoke used in venues.
Aajonus said that another musician he worked with, who was in a show that used heavy smoke for two hours across an entire year, developed a lesion on the neck that began draining about three months after the musician quit the show. The drainage he described as similar to liquid smoke coming out through a small hole. The topical treatment he applied in that case was fresh pineapple and vinegar, alternating. He specified fresh pineapple: a slice rubbed onto the area.
Iridology and Secondhand Tar Accumulation
In multiple seminar body readings, Aajonus identified secondhand smoke damage through iris analysis and hand reading. He described what the tars look like in the iris: in direct smokers, concentrated spots around the lung and kidney zones; in secondhand smokers, patches distributed throughout the iris, appearing in multiple organ zones without the deep focal concentration of a direct smoker.
He described one reading where the person had never smoked but had been raised around smoking parents, and he identified the pattern immediately as secondhand smoke because it was in particular spots throughout the system rather than concentrated like a direct smoker's accumulation. He also found tar deposits in unusual locations such as the esophagus all the way down to the stomach, throughout the left kidney, and extensively in the brain for individuals who had long-term secondhand exposure.
He noted in one reading that some of the tar accumulation appeared fresher, meaning more recent, while other accumulation appeared older. In one case he mentioned that some of the fresher material seemed to have passed from the person's mother, indicating that prenatal or early childhood exposure could introduce tars that remain identifiable decades later. He also observed that campfires and oil paint fumes could produce similar-looking accumulations in the iris, complicating identification when multiple sources of exposure were present.
How Bodies Process Smoke Tars
Aajonus described two different responses he had observed, which he acknowledged to be contradictory in appearance. In most individuals, tars accumulate and harden over years, causing progressive tissue damage. However, he noted that a small number of people on the diet for many years showed no visible tar accumulation even though they continued to smoke. He described two people who had been on the Primal Diet for sixteen years and still smoked, and who showed no tars detectable in the body. His interpretation was that some individuals are able to convert the tars into a usable fat and utilize them without toxic accumulation. He acknowledged not knowing how many smokers have this capacity, but observed it was rare.
For everyone else, tars accumulate and must be actively eliminated. He described the body's preferred elimination routes: out through the skin when the body is strong enough and has adequate fat to buffer the process, and through mucus drainage when cleansing begins. He described his own experience of smelling and tasting smoke toxins during a detoxification period years after he had stopped smoking, identifying it as the tars from age 8 to 24 breaking down. He noted that in his case the tars came out through the skin rather than through the lungs, and interpreted this as the body choosing a safer elimination route: "Instead of in my lungs, it did it in the skin, where it could handle it easier. If I had to do that in the lungs, my lungs would have been filling with water."
The body's ability to initiate this elimination is dependent on available raw fats to act as solvents and buffers. Without sufficient raw fat, the tars remain stored and continue to crystallize.
Protocols for Dissolving Smoke Tars
Aajonus developed specific protocols for dissolving crystallized smoke tars, and he specified that these apply to secondhand smokers as much as to direct smokers. His primary remedy was green tobacco juice, fermented with raw apple cider vinegar and lemon juice.
**Green Tobacco Juice**
The tobacco must be green, not dried or cured. He explained that cured tobacco is the taxed form, while green tobacco is the raw plant. It grows in a humid climate and is seasonal, typically available from July through September, sometimes as late as mid-September in some years. He advised not contacting sources outside the growing season.
The preparation process: juice the green tobacco through a Green Star or similar masticating juicer until 12 or 30 ounces of juice have been obtained. The smaller quantity (12 ounces) is for light smokers and secondhand smokers. The larger quantity (30 ounces) is for heavy long-term smokers. Keep the juice in a glass jar.
To each serving: add 1 ounce of good water (from a well or glass-bottled, if carbonated then shaken until the carbonation dissipates) for light and secondhand smokers, or 2 ounces for heavy smokers. Add 3 drops of raw apple cider vinegar for light and secondhand smokers, or 6 drops for heavy smokers. Add 3 drops of lemon juice for light and secondhand smokers, or 6 drops for heavy smokers. Blenderize for 20 seconds, then allow to ferment in a warm place for 3 to 7 weeks before using.
Dosage: light and secondhand smokers take 3 drops per day, which will consume 12 ounces over 9 months. Heavy long-term smokers take up to 7 drops per day, consuming 30 ounces over 9 months. Aajonus specified that drops of the juice are added to a vegetable juice for consumption.
He addressed the question of whether tobacco turning brown after harvesting would compromise the remedy. His answer was that if it were brown for several days, fermentation would still work correctly. The requirement for green tobacco was simply to ensure that it was not the dried, cured form. He also mentioned that chewing raw green tobacco and spitting out the pulp could help dissolve tars, and that even allowing chewed tobacco to mold and then consuming it could assist with breakdown. He said he had never heard of anyone becoming nauseous from raw green tobacco, in contrast to the reaction from processed or cured tobacco products.
**Vegetable Juice**
For heavy secondhand smoke accumulation throughout the system, he recommended a specific vegetable juice formula: approximately 25% celery, 10% spinach, 15% coriander leaves and stalks, 20% cucumber puree, and the remainder carrot. The green tobacco drops would be added to this juice. He also recommended pineapple extensively, stating that it helps break down tar, and any kind of citrus.
**Vinegar**
Aajonus mentioned vinegar as a dissolver of smoke accumulation in multiple contexts. He told one person that vinegar would help dissolve the patched tar accumulation characteristic of secondhand exposure "a little bit." He also included vinegar in his sport formula for people with nightclub tar deposits in the bronchioles and lungs: a tablespoon of vinegar with three to four ounces of milk, once every other day around six o'clock.
**Topical Application for External Discharge**
When tars were discharging through the skin or through a tumor site, Aajonus recommended topical applications of fresh pineapple and vinegar alternated on the area. He described rubbing a slice of fresh pineapple directly on the skin surface. For Malcolm Gold's eye tumor, he used lime juice and vinegar. He described the tumor shrinking and ultimately resolving without medical intervention using this protocol.
**Butter, Cream, and Coconut Oil for Smoke Inhalation**
For acute smoke inhalation exposure, such as during the Malibu fires Aajonus personally survived, he recommended butter and lubrication formulas internally to protect the tissues from the smoke damage. He confirmed that coconut oil or olive oil added to the protocol would also assist. He described this as helping with "any internal smoke inhalation."
**Meat Balance**
For secondhand smokers identified through iridology, Aajonus recommended a dietary balance of 50% red meat and 50% white meat rather than heavy reliance on red meat alone. He suggested that combining red meat with egg would bring it to a better balance. He also noted that proteins in general are needed to bind with carbon monoxides and tars, and help break them down into solvents the body can use. Insufficient protein while being exposed to smoke or while detoxifying from it will cause more sensitivity to pain and slower breakdown of the accumulated tars.
The Mechanic's Indoor Exposure
One case Aajonus described involved a mechanic who had worked for years in a shop where smoking was common. In reading this person, Aajonus found that the body held smoke damage at a level nearly indistinguishable from direct smoking. He noted that in addition to the tobacco smoke, the mechanic was also exposed to brake fluid and other chemical compounds that irritated the tissues and would cause bile accumulation throughout the body. The chlorine vapors from chloroform in the environment might also be contributing to the lung tissue's tendency to hold others' smoke more deeply. He told the person directly that being around smoking workers made him a smoker too, regardless of whether he personally ever held a cigarette.
Guilt and Choice Position
Aajonus consistently stated that individuals should not feel guilty about their exposure to smoke, whether direct or secondhand. He stated that for secondhand exposure specifically, the person often had no choice, particularly in childhood. His role was to identify what was in the body and address it practically, not to assign blame. For people actively smoking who could not stop, he offered the honey and butter protocol before each cigarette craving rather than demanding cessation, and he said he would never tell someone to stop smoking without first addressing the underlying blood sugar and enzyme need that the nicotine rush was temporarily satisfying. The same non-judgmental framework applied to secondhand smokers, who accumulated damage through circumstances often beyond their control.
