Neurons

Each neuron has a central cell body with numerous projections called neurites. At the end of each neurite is a growth cone, the site where structural proteins are assembled into the cell membrane. Two proteins are principal to this process. Actin is responsible for the pulsating motion visible in growing neurons. Tubulin is the major structural component of the neurite membrane itself. During normal cell growth, tubulin molecules link together end-to-end to form microtubules, which surround another structural protein component called neurofibrils inside the neuronal axon.

Aajonus drew on research from the University of Calgary's Faculty of Medicine to illustrate how neurons grow and how that growth is disrupted. In their tissue cultures, neurons from snail brain tissue displayed linear growth driven by growth cone activity. He emphasized that growth cones in all animal species, from snails to humans, have structurally and behaviorally identical characteristics and use proteins of virtually identical composition. This cross-species uniformity made the snail tissue a valid model for understanding what happens to human neurons.

Aajonus stated this repeatedly because the conventional understanding blurs the distinction between neurons and nerves. Nerves, which register pain, run through the body and through the meningi surrounding the brain, but the brain's internal tissue is made of neurons. Neurons do not register pain. This is why, as Aajonus noted, the film Hannibal shows a character eating another person's brain while that person remains awake and conversational, because the brain itself cannot feel the extraction. The meningi, with its up to eleven layers of distinct tissue, each layer slightly different in composition, is where the experience of pain originates in the head.

The ganglia, synapse, axons, and all the associated structures of the neuron network can be reproduced and rebuilt. The condition is raw meat in the diet.

Mercury is the most significant neurotoxin Aajonus discussed in the context of neurons, and the evidence he returned to most often was a video produced by the University of Calgary, also referenced as Alberta University and the University of Toronto in different transcripts. The video, a time-lapse recording of neurons growing in a petri dish, shows what happens when a 2% solution of thimerosal, the liquid mercury compound used in vaccines, eye drops, and wound treatments such as mercurochrome and methylate, is introduced.

Upon exposure, the neurons do not merely slow their growth. They begin dissolving and disappearing. Aajonus described it as watching them disintegrate and evaporate. He also described a variation where the mercury was not placed directly onto the neurons but positioned at a distance from the petri dish. Even without direct contact, within seconds, the neurons began disintegrating. The vapor alone was sufficient to destroy them.

The University of Calgary research specified the mechanism. Tubulin proteins link together to form microtubules that support the neurite structure. When mercury ions enter the culture medium, they infiltrate the cell and bind to newly synthesized tubulin. Because bound GTP normally provides the energy that allows tubulin molecules to attach to one another, mercury ions disrupting that binding cause the microtubule assembly to fail. The neurite microtubules begin to disassemble. The result is that the developing neurite structure collapses, and denuded neurofibrils form aggregates or tangles. In the Calgary experiments, neurons isolated from snail brain tissue were grown in culture for several days and then exposed to very low concentrations of mercury for just twenty minutes. Over the subsequent thirty minutes, the neurite membrane underwent rapid degeneration, leaving behind only the exposed neurofibrils.

Crucially, other heavy metals added at the same concentration, including aluminum, lead, cadmium, and manganese, did not produce this effect. Mercury is specifically destructive to neurite membrane structure in a way that distinguishes it from all other heavy metals tested.

Aajonus connected this to the thimerosal content of vaccines. He stated that a 2% thimerosal solution contains approximately 76 quadrillion molecules of mercury. The mercury a person receives in a single vaccine is not comparable to what might be present in fish. The mercury in fish represents less than a fraction of a hundredth of a percent of what is delivered in one vaccine injection. Children in Illinois, he noted, were receiving approximately 128 vaccines by the age of sixteen. In laboratory tests, only 7% of injected mercury was found to be excreted through the urinary tract within the first 24 hours, and 24% through another route, meaning the vast majority remains in the body.

Aajonus reported that mercury went into his own communication center as a child and made him autistic. He had spoken his first word at five and a half months, among the earliest in his family. After his third tetanus shot, that changed entirely.

Aajonus distinguished between what mercury does to neurons and what aluminum does, treating them as producing distinct and recognizable patterns of neurological impairment.

Mercury fragments and destroys neuron cells outright. If a person has lost memories permanently and cannot retrieve something at all, that is typically a mercury problem, because mercury destroys the neuron structure itself. The loss is permanent because the physical substrate of the memory has been disintegrated.

Aluminum does not destroy neurons but disrupts their function by attacking the zeta potential. The zeta potential is the fluid's ability to keep nutrients suspended. Aajonus illustrated this with an aquarium analogy: put liquid aluminum into aquarium water and the fish hit the bottom and can no longer swim. In the neurological system, the same thing happens when aluminum is present in the fluid through which the synapse fires. Rather than the synapse rocketing information through the axons and ganglia to the rest of the brain, it fires and the signal simply slides down and sits in the dendrites and the connective veins and arteries between. The thought does not complete its journey. The experience is a thought that vanishes before arriving, a name that cannot be retrieved even though it is not permanently gone. Aajonus described this from personal experience as a child, when no complete thought on language ever arrived. He pointed to carrot juice and the carotene in it as something that may bind with and neutralize aluminum, which he credited with beginning to change his own neurological function.

The neurological fluid is the thinnest of the body's three fluid systems. It transmits light and conducts electricity. Its primary functional components are metallic minerals. Aluminum, mercury, lead, cadmium, arsenic, thallium, zinc, iodine, and other trace metals are all present in this fluid in very small amounts and serve the neurological system's operations.

The metallic minerals found in trace amounts in all natural foods are sufficient to run the nervous system when the food is consumed raw and the ionic bonds are intact. The problem arises when food is cooked. Cooking fractures the ionic bonds between these minerals and their associated nutrients. Once the bonds are broken, the metals become free radicals. As electromagnetic particles without their nutrient partners, they congregate or repulse each other based on electromagnetic charge rather than bioactive chemistry. They are no longer functional in the same way.

Because the brain and nervous system are the primary users of metallic minerals in the body, and because fat is where the body stores toxins, and because the brain and nervous system are 60 to 80 percent fat, the free radical metallic minerals released by cooking, industrial pollution, vaccines, and environmental contamination flow predominantly into the brain and nervous system. The concentration of free radical metals in the human brain is, according to Aajonus's reading of the data, 1,200,000 times the concentration found in any other animal's brain. No other animal has a toxic brain in the way humans do.

Beyond metallic contamination, Aajonus described a separate mechanism by which neurons are impaired: advanced glycation end products (AGEs) produced when high carbohydrate intake, particularly in the early part of the day, floods the neurological system with sugar. When the body runs on high carbohydrates, it produces AGEs in large quantities. In the neurological fluid, AGEs cause stickiness. The neurons, axons, ganglia, and synapse structures all depend on fluid that allows clean transmission. When the neurological fluid becomes sticky from AGE buildup, synapse firing misfires. The ganglion and axon do not pulsate the same way. Signals go to the wrong places, bounce incorrectly, and produce confused or slow thinking.

By contrast, when the body makes its glycogen from meat using pyruvate as the intermediary rather than from direct carbohydrate conversion, only approximately 7% advanced glycation end product results. This keeps the neurological fluid clean enough for proper synapse transmission. The practical implication was that meat in the early part of the day, rather than high-carbohydrate foods, preserves the clarity and speed of neural signaling throughout the day.

Aajonus was explicit that the medical community's position that neurons cannot regenerate is wrong, and that his direct experience with animals and humans contradicted it. In his laboratory tests with animals, raw meat was the only substance that helped regenerate nerve tissue. Cooked food, regardless of protein content, did not produce regeneration. The chemistry produced by cooked food, combined with its toxicity load, renders the conditions for neuronal division impossible.

Animal cells reproduce by dividing: a cell grows, divides, and the resulting cells grow and divide again. Neurons are capable of this process. The capacity is suppressed not by inherent biological limitation but by the toxic and nutritionally degraded environment produced by a cooked food diet. With raw meat, ganglia, synapse, and neurons can all be reproduced and rebuilt.

Aajonus argued that the enlargement of the human brain over evolutionary and historical time is not evidence of increasing intelligence but of increasing toxicity. When Lucy, the early hominid, was living, she had a brain roughly half the size of the modern human brain, and she did not cook food. The brain began to enlarge when humans started smelting metals, mining rocks, burning coal, and cooking food at greater and greater scales. Each of these activities released metallic vapors and free radical toxins that went into the brain. The brain had to expand its fat content to store all the incoming toxins and protect the rest of the brain tissue from them.

Einstein used only 12% of his brain. If a genius uses 12%, what is the remaining 88% doing? Aajonus's answer was that it is storing toxins. The brain functions as a storage depot for metallic and industrial toxins because its high fat content makes it the body's primary toxin reservoir. A bigger brain in the modern human is not a smarter brain; it is a more toxic brain that has expanded its capacity to contain the poisons the body cannot eliminate.

The concentration of these free radical metals in the neurological fluid disrupts all downstream neurological functions. Thoughts fail to complete. Memory is impaired or destroyed depending on which metal is dominant. Language, motor coordination, emotional regulation, and the entire spectrum of neurological activity is compromised when the medium through which neurons communicate is contaminated.

Aajonus identified specific foods as essential for rebuilding and supporting neurological tissue and neuron function.

White meat of fowl, particularly chicken and turkey, was the primary food for rebuilding the myelin sheath and the neurological framework more broadly. His experiments with animals showed that those fed raw chicken consistently had better neurological structure, better skin, better coats, and better intestinal integrity compared to those fed other meats. Fish could help replenish neurological fluids, and shellfish and oysters were also identified as supportive. However, fish alone did not rebuild the myelin or restore neurological progression. Without fowl, the nervous system did not rebuild as well. Aajonus noted this was a correction from what he had stated in his first book, where he said fish could restore myelin. His later experiments showed that was incomplete.

For the brain and nervous system to receive fat, a specific delivery mechanism was needed because dietary fats are typically absorbed before food reaches the colon. Aajonus described a method involving the introduction of fats such as cream directly through the intestinal route, combined with specific body movements to move the substance across the transverse colon and down toward the ascending colon. By this mechanism, fresh fats that the brain and nervous system would otherwise never receive could be delivered directly. When the myelin receives adequate fat this way, a noticeable shift occurs in nervous system function.

Meat-derived glycogen, produced through pyruvate rather than through carbohydrate conversion, keeps the neurological fluid free of the stickiness that prevents proper synapse firing.